SELTA is a TLR2/6 agonist derived from the normal microbial flora of a healthy
individual, suggesting the potential for limited off-target effects and
toxicity.
Downstream TLR2/6 signaling after SELTA leads to anti-inflammatory and analgesic effects.
Well-defined path to proof of concept:
- Tailored drug formulation approaches to target the needs of specific disease
populations. - Unique MOA, with preclinical data demonstrating potential clinical
applications across multiple autoimmune and chronic inflammatory diseases.
Potential to be first-in-class molecule targeting TLR2/6 for auto-immune
diseases.